BCL2L13
BCL2L13proteinBCL2L13 suppression by miR-96-5p promotes cardiomyocyte apoptosis during myocardial ischemia and infarction.
Pathway placement
Cascade stepMyocardial injury (shared endpoint)
Confidencemedium
RationaleAnti-apoptotic protein; miR-96-5p target regulating cardiomyocyte survival.
Druggability
DruggableYes
Known drugs / candidates0
Small-molecule tractableNo
Antibody tractableYes
EnsemblENSG00000099968
Assay & specimen
Class-level default (no specific cleared assay)— generic method inferred from analyte class; confirm against a specific product insert before use.
Specimen
Serum or plasma
Collection tube
Serum separator (gold/red-top, SST) · K2/K3-EDTA (lavender-top)
Method / principle
Sandwich immunoassay (ELISA) — research-grade unless a cleared assay exists
Reagent / substrate
Matched anti-target antibody pair (capture + labeled detection)
Platform
ELISA microplate or multiplex (Luminex/MSD)
Turnaround · availability
Send-out / research · Research-grade (no universal clinical assay)
Literature evidence(1)
- Serum miR-96-5p is a novel and non-invasive marker of acute myocardial infarction associated with coronary artery disease.Bioengineered · 2022 · PMID 35109756 · doi