Interleukin-17E
IL17FproteinIL-17E drives vascular inflammation and predicts post-MI cardiac remodeling through pro-inflammatory T-cell signaling.
Pathway placement
Cascade stepPlaque inflammation
Confidencemedium
RationaleInflammatory mediator in CAD; IL-17 family promotes plaque inflammation and destabilization.
Also acts inPlatelet activation
Druggability
DruggableYes
Known drugs / candidates2
Small-molecule tractableYes
Antibody tractableYes
EnsemblENSG00000112116
Type I vs Type II discrimination
ScoresLow-confidence (proxy)
R — rupture / Type-I26
C — confounder / Type-II49
A — assay feasibility72
E — evidence strength19
T1DI (composite)5
Specificity differential (R−C)-22.8
Confounder panel (Type-II drivers)
No confounder evidence retrieved.
Tier: light (literature co-occurrence proxy — lower confidence). See the discrimination table for all markers.
Assay & specimen
Class-level default (no specific cleared assay)— generic method inferred from analyte class; confirm against a specific product insert before use.
Specimen
Serum or plasma
Collection tube
Serum separator (gold/red-top, SST) · K2/K3-EDTA (lavender-top)
Method / principle
Sandwich immunoassay (ELISA) — research-grade unless a cleared assay exists
Reagent / substrate
Matched anti-target antibody pair (capture + labeled detection)
Platform
ELISA microplate or multiplex (Luminex/MSD)
Turnaround · availability
Send-out / research · Research-grade (no universal clinical assay)
Literature evidence(3)
- Comprehensive Quality Analysis of Conventional and Novel Biomarkers in Diagnosing and Predicting Prognosis of Coronary Artery Disease, Acute Coronary Syndrome, and Heart Failure, a Comprehensive Literature Review.Journal of cardiovascular translational research · 2024 · PMID 38995611 · doi
- Harnessing the Plasma Proteome to Mirror Current and Predict Future Cardiac Remodeling After Myocardial Infarction.Journal of cardiovascular translational research · 2022 · PMID 36197585 · doi
- Neutrophils, neutrophil extracellular traps and interleukin-17 associate with the organisation of thrombi in acute myocardial infarction.Thrombosis and haemostasis · 2012 · PMID 23238559 · doi