12(S)-HETE
lipidProinflammatory eicosanoid mediating leukocyte recruitment and vascular inflammation post-MI.
Pathway placement
Cascade stepPlaque inflammation
Confidencehigh
RationaleLipoxygenase eicosanoid mediating proinflammatory response post-MI.
Druggability
Not assessed (no mapped human gene target).
Type I vs Type II discrimination
ScoresLow-confidence (proxy)
R — rupture / Type-I32
C — confounder / Type-II38
A — assay feasibility40
E — evidence strength16
T1DI (composite)3
Specificity differential (R−C)-6.4
Confounder panel (Type-II drivers)
No confounder evidence retrieved.
Tier: light (literature co-occurrence proxy — lower confidence). See the discrimination table for all markers.
Assay & specimen
Class-level default (no specific cleared assay)— generic method inferred from analyte class; confirm against a specific product insert before use.
Specimen
Serum or plasma (EDTA to limit oxidation)
Collection tube
K2/K3-EDTA (lavender-top) · Serum separator (gold/red-top, SST)
Method / principle
LC-MS/MS lipidomics (targeted or shotgun)
Reagent / substrate
Deuterated lipid-class internal standards; MS/MS transitions
Platform
LC-MS/MS
Turnaround · availability
Research · Research-only
Literature evidence(2)
- Obesity superimposed on aging magnifies inflammation and delays the resolving response after myocardial infarction.American journal of physiology. Heart and circulatory physiology · 2014 · PMID 25485899 · doi
- Biomarkers in the early period of acute myocardial infarction in rat serum and protective effects of Shexiang Baoxin Pill using a metabolomic method.Journal of ethnopharmacology · 2011 · PMID 22001859 · doi