LysoPC(18:2(9Z,12Z))
lipidLysophosphatidylcholine species altered in acute coronary syndrome, marking lipoprotein oxidation and inflammatory lipid-mediator activation.
Pathway placement
Cascade stepLipid retention & oxidation
Confidencemedium
RationaleLipid mediator; inversely correlates with atherosclerosis burden; involved in lipoprotein remodeling.
Also acts inVascular inflammation, Myocardial injury
Druggability
Not assessed (no mapped human gene target).
Assay & specimen
Class-level default (no specific cleared assay)— generic method inferred from analyte class; confirm against a specific product insert before use.
Specimen
Serum or plasma (EDTA to limit oxidation)
Collection tube
K2/K3-EDTA (lavender-top) · Serum separator (gold/red-top, SST)
Method / principle
LC-MS/MS lipidomics (targeted or shotgun)
Reagent / substrate
Deuterated lipid-class internal standards; MS/MS transitions
Platform
LC-MS/MS
Turnaround · availability
Research · Research-only
Literature evidence(3)
- Metabolic Profiling Reveals Diagnostic Biomarkers for Distinguishing Myocarditis From Acute Myocardial Infarction.Cardiovascular therapeutics · 2025 · PMID 40270589 · doi
- Multinomial machine learning identifies independent biomarkers by integrated metabolic analysis of acute coronary syndrome.Scientific reports · 2023 · PMID 37996510 · doi
- Metabolomic characterization of myocardial ischemia-reperfusion injury in ST-segment elevation myocardial infarction patients undergoing percutaneous coronary intervention.Scientific reports · 2019 · PMID 31409856 · doi