Jun proto-oncogene, AP-1 transcription factor subunit
JUNgeneJUN orchestrates inflammatory gene programs destabilizing atherosclerotic plaques.
Pathway placement
Cascade stepPlaque inflammation
Confidencehigh
RationaleInflammatory transcription hub; regulates immune activation and vascular remodeling.
Also acts inCap degradation / rupture, Endothelial activation/erosion
Druggability
DruggableYes
Known drugs / candidates0
Small-molecule tractableYes
Antibody tractableNo
EnsemblENSG00000177606
Type I vs non-Type I discrimination
ScoresShared / rises in both
R — rupture / Type-I67
C — non-Type-I53
A — assay feasibility52
E — evidence strength59
T1DI (composite)17
Specificity differential (R−C)+13.4
Non-Type-I axis panel
Does this marker also move in each non-Type-I setting? mag 0–3; higher means less Type-I-specific. n/a = no evidence retrieved, which is not the same as no change.
2sepsis / systemic inflammationn/a
2anemia / acute blood lossn/a
2hypovolemia / dehydrationn/a
2tachyarrhythmian/a
2hypoxemia / respiratory failuren/a
2hypertensive emergencyn/a
2high-demand / peri-operative stressmag 2
3sudden cardiac deathmag 1
4aPCI-related periproceduraln/a
4bstent thrombosismag 1
4cin-stent restenosismag 2
5CABG-relatedmag 2
Coverage: 5/12 axes with evidence
Tier: deep-scored (abstract-extracted) · 9 supporting references. See the discrimination table for all markers.
Assay & specimen
Class-level default (no specific cleared assay)— generic method inferred from analyte class; confirm against a specific product insert before use.
Specimen
Whole blood — gene is not a circulating analyte; measure protein product or genotype
Collection tube
K2-EDTA whole blood (lavender-top)
Method / principle
SNP genotyping / sequencing; or immunoassay of encoded protein
Reagent / substrate
Allele-specific primers/probes (TaqMan) or NGS panel; or antibody for protein
Platform
qPCR / NGS / array
Turnaround · availability
Send-out · Genotyping widely available; protein assay variable
Literature evidence(3)
- Identification of biomarkers associated with coronary artery disease and non-alcoholic fatty liver disease by bioinformatics analysis and machine learning.Scientific reports · 2025 · PMID 39875572 · doi
- Single-cell transcriptomic analysis of peripheral blood mononuclear cells reveals key immune responses in ST-segment elevation myocardial infarction.Hereditas · 2025 · PMID 41291938 · doi
- Integrated analysis of gene expression changes associated with coronary artery disease.Lipids in health and disease · 2019 · PMID 30961613 · doi